Medicine:Viral cardiomyopathy
| Viral cardiomyopathy | |
|---|---|
| Specialty | Cardiology |
| Symptoms |
|
| Causes | viral infections cause myocarditis |
| Diagnostic method | Electrocardiography, Echocardiography, MRI[1] |
Viral cardiomyopathy occurs when viral infections cause myocarditis with a resulting thickening of the myocardium and dilation of the ventricles. These viruses include Coxsackie B and adenovirus, echoviruses, influenza H1N1, Epstein–Barr virus, rubella (German measles virus), varicella (chickenpox virus), mumps, measles, parvoviruses, yellow fever, dengue fever, polio, rabies and the viruses that cause hepatitis A and C,[2][3][4] as well as COVID-19,[5][6] which has been seen to cause this in persons otherwise thought to have a "low risk" of the virus's effects.[7]
COVID-19 Cardiomyopathy
Patients with Covid-19 frequently experience heart issues.[8] According to studies, people who have had previous cardiovascular conditions like cardiomyopathy, hypertension, coronary heart disease, or arrhythmia are more likely to become critically ill from SARS-CoV-2 infection. Myocarditis may result from a direct viral infection of the myocardium.[8] Cardiovascular biomarkers like troponin, lactate dehydrogenase, high sensitivity amino-terminal B-type natriuretic peptide, creatinine kinase, and creatinine kinase myocardial band, which indicate myocardial damage, increase in concentration in response to covid-19.[8] Hundreds of studies have reported myocarditis/myopericarditis caused by Covid-19 infection in living patients, with a male predominance (58%), and a median age of 50 years. [8]
See also
References
- ↑ Schultz, Jason C.; Hilliard, Anthony A.; Cooper, Leslie T.; Rihal, Charanjit S. (1985-01-01). "Diagnosis and Treatment of Viral Myocarditis". Mayo Clinic Proceedings 84 (11): 1001–1009. doi:10.1016/S0025-6196(11)60670-8. PMID 19880690.
- ↑ "A case series of reversible acute cardiomyopathy associated with H1N1 influenza infection". Methodist DeBakey Cardiovascular Journal 8 (1): 42–5. Jan 2012. doi:10.14797/mdcj-8-1-42. PMID 22891110.
- ↑ Badorff C; Lee G. H.; Knowlton K. U. (2000). "Enteroviral cardiomyopathy: bad news for the dystrophin-glycoprotein complex.". Herz 25 (3): 227–32. doi:10.1007/s000590050011. PMID 10904843.
- ↑ "A rare case of Epstein-Barr virus-induced dilated cardiomyopathy". Heart & Lung 40 (1): 81–7. 2011. doi:10.1016/j.hrtlng.2009.12.012. PMID 20561866.
- ↑ Rathore, Sawai Singh; Rojas, Gianpier Alonzo; Sondhi, Manush; Pothuru, Suveenkrishna; Pydi, Reshma; Kancherla, Neeraj; Singh, Romil; Ahmed, Noman Khurshid et al. (2021). "Myocarditis associated with Covid-19 disease: A systematic review of published case reports and case series" (in en). International Journal of Clinical Practice 75 (11): e14470. doi:10.1111/ijcp.14470. ISSN 1742-1241. PMID 34235815.
- ↑ Siripanthong, Bhurint; Nazarian, Saman; Muser, Daniele; Deo, Rajat; Santangeli, Pasquale; Khanji, Mohammed Y.; Cooper, Leslie T.; Chahal, C. Anwar A. (September 2020). "Recognizing COVID-19–related myocarditis: The possible pathophysiology and proposed guideline for diagnosis and management". Heart Rhythm 17 (9): 1463–1471. doi:10.1016/j.hrthm.2020.05.001. ISSN 1547-5271. PMID 32387246.
- ↑ "Recognizing COVID-19–related myocarditis: The possible pathophysiology and proposed guideline for diagnosis and management". Heart Rhythm 17 (9): 1463–1471. 5 May 2020. doi:10.1016/j.hrthm.2020.05.001. PMID 32387246. PMC 7199677. https://www.heartrhythmjournal.com/article/S1547-5271(20)30422-7/fulltext.
- ↑ 8.0 8.1 8.2 8.3 Razaghi, Ali; Szakos, Attila; Al-Shakarji, Riham; Björnstedt, Mikael; Szekely, Laszlo (2022-12-31). "Morphological changes without histological myocarditis in hearts of COVID-19 deceased patients". Scandinavian Cardiovascular Journal 56 (1): 166–173. doi:10.1080/14017431.2022.2085320. ISSN 1401-7431. PMID 35678649.
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